IL - 1 , IL - 6 , and TNF - ain cachexia patients with different syndromes were examined.
同时,对现在住院的患者选择不同辨证分型的恶病质病人检测IL -1 、 IL -6, TNF - α.
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Serum IL 6 and sIL 6 R were measured by Elisa assay inall subjects.
采用Elisa法测定3组的血清IL-6、sIL-6R浓度.
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There were significant differences between two groups in menopause duration , ALP, BGP, BMD, and IL - 6 ( P & gt; 0.05 ).
绝经年限、血总碱性磷酸酶(ALP) 、 血清骨钙素(BGP) 、 骨密度 、 血浆IL -6 浓度有显著性差异 ( P & lt; 0.05 ).
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GCF IL - 6 was measured by 125 I radioimmunoassay ( RIA ) .
应用放射免疫分析法检测 GCF 中IL - 6的含量.
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Clinical parameters serum calcium had significant difference between normal group and abnormal group about IL - 6 ( P < 0.05 ).
血钙正常组和异常组间的IL-6水平存在显著不同 ( P<0.05 ).
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Objective To investigate the clinic value of interleukin - 6 ( IL - 6 ) in sepsis and septic shock.
目的探讨白细胞介素 -6 ( IL -6 ) 在新生儿败血症及败血症休克中的变化及临床意义.
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Objective : To investigate the significance of plasma tumor necrosis factor - α ( TNF - α ) and interleukin - 6 ( IL - 6 ) levels in acute poisoning.
目的: 研究急性中毒患者血清肿瘤坏死因子 - α ( TNF - α ) 和白细胞介素-6 ( IL -6 ) 的水平及其临床意义.
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Purpose To investigate the relationship between serum interleukin 6 ( IL 6 ), soluble interleukin 6 receptor ( sIL 6 R ) levels and low T 3 syndrome.
目的研究白介素6 ( IL-6 ) 和可溶性白介素6受体 ( sIL-6R ) 与低T3综合征的关系.
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Serum NSE was measured by solid phase ethanol immunoadsorption and serum IL - 6, by double antibody ELISA.
以固相醇联免疫吸附法(SPEIA)测定血清NSE水平,以双抗体夹心ELISA法测定血清IL - 6水平.
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Conclusion C . pneumoniae rHsp 10 can induce the secretion of TNF - α and IL - 6 by mu - rine macrophages.
结论表达并纯化的rHsp10蛋白能诱导MΦ分泌TNF - α和IL -6 等促炎因子.
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Purpose: Investigating the effect of IL - 6 transgenic expression on the negative growth regulation of colon cancer cells.
目的: 将外源性IL-6 基因导入大肠癌细胞,建立IL-6 基因表达株,观察IL -6 转基因表达对大肠癌细胞的体内外抑制作用.
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Meanwhile serum IL - 6 , IL - 18 and CRP level were also elevated in patients with ACS.
同时ACS患者 血清IL -6 、 IL -18 、CRP水平明显高于SA及正常对照组.
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Serum level of IL - 6 in PRA was significantly higher than that of healthy controls ( PO .05 ).
PRA患者血清IL -6 水平高于健康对照组 ( P<0.05 ).
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CONCLUSION: LPS induces liver IL - 6 expression , which might activate BEC proliferation through STAT 3 pathway.
结论: LPS刺激导致肝组织IL-6分泌增加, 后者可能通过STAT3介导BEC增殖.
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Results: PCT 、 CRP and IL - 6 blood level increased significantly in severe infection disease, but PCT increased more signifi - cantly ( P & lt; 0.01 ).
结果: 重症感染时,血清PCT、CRP和IL -6 浓度升高均有显著性, 但PCT显著性更高 ( P & lt; 0.01 ).
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