K 562 , a human erythroleukemia cell line, was widely used.
K562为人的红白血病细胞系,常被用于白血病诊断 、 疗的俐晰究.
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Objective : To observe the apoptosis - inducing effects of dopamine on K 562 leukemia cells.
目的: 观察多巴胺对白血病细胞系K562的诱导凋亡作用.
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Conclusion: ACV can inhibit proliferation and induce erythrodifferentiation of K 562 cells.
结论: ACV对K562细胞具有抑制增殖和诱导分化作用,该结果可为慢性粒细胞白血病的治疗探索新的途径.
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A 23187 might accelerate the transformation of K 562 cells into killer cells.
IL-15 可提高K562细胞向杀伤细胞转化的诱导率;A23187可能加速K562细胞向杀伤细胞的转化.
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Dihydroarteannuin inhibited K 562, KB, HCT , SGC 7901 , H ela, HepG, BGC 823, KCC 853, Bel 7402, A 549, B 16 - BL 6 cells.
双氢青蒿素对K562 、 KB 、 HCT、SGC7901 、 Hela、HepG 、 BGC823 、 KCC853 、 Bel7402 、 A549 、 B16-BL6细胞均有抑制作用.
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Conclusion Wortmannin could inhibitedtheproliferationofK 5 6 2 cells, andwascellcyclespecificagent ( CCSA ).
作用24h后 细胞实验组细胞BCL-2 蛋白表达较对照组降低, 说明 Wortmannin 可促进K562细胞的凋亡.结论 Wortmannin可以 通过抑制PI3K通路 抑制K562细胞的增殖.
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To study the possibility and the mechanism of K 562 apoptosis induced by microwave and etoposide.
探讨微波和足叶乙苷体外诱导K562细胞凋亡的可能性及其机制.
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Objective : To study the mechanism of apoptosis induced by Orindonin in K 562 cells.
目的: 研究冬凌草甲素诱导白血病K562细胞凋亡的机制.
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Results Apoptosis of K 562 cells was significantly increased associated with inhibition of bcr abl expression.
结果bcr?abl融合基因表达受抑后,K562细胞凋亡显著增加.
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Research furtherly the CTLs killing ability in vitro to GLC - 82, CALU - 6 and K 562 cell.
进一步探讨该CTL对 GLC-82, 肺癌CALU -6 和人红白血病K562细胞的体外杀伤效应.
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Research furtherly the CTL ? ? s killing ability in vitro to GLC - 82, CALU - 6 and K 562 cell.
进一步探讨该CTL对GLC-82, 肺癌CALU -6 和人红白血病K562细胞的体外杀伤效应.
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Meanwhile, it could induce apoptosis obviously and up - regulate the levels of Fas in K 562 cells.
并能诱导K562细胞凋亡,使Fas蛋白水平上调.
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Conclusion Prazosin can inhibit the proliferation of K 562 cells and induce the apoptosis of K 562 cells.
结论:哌唑嗪能抑制K562白血病细胞的增殖,并能诱导其凋亡.
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Methods: K 562 cells were cultured with ACV for 4 d to detect cellular changes.
方法: 将无环鸟苷加入K562细胞培养4d,测定其活细胞数目、联苯胺染色阳性率,光镜观察形态、组织化学染色.
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