Prolonged uremia significantly alters the ligand - binding properties of albumin, and albumin contributeswhole blood acid - base buffering capacity.

持续的尿毒症明显改变了白蛋白的配体结合特性, 而白蛋白也与血液酸碱缓冲能力有关.

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Consequent upon the structural flexibility and the ligand binding, circulating albumin exhibits chemical and physical microheterogeneity.

由于其结构上的易变性和配体结合的不同, 循环中的白蛋白表现出化学和物理上的微观不均一性.

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