HSF 1 function increases survival under a great many pathophysiological conditions.
在许多病理生理条件下,HSF-1 的功能增加了(个体)存活.
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CONCLUSION: IGF - 1 takes part in the pathophysiological procedure of acute cere bral infarction.
结论: IGF -1 参与了急性脑梗死的病理生理过程.
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The main pathophysiological changes of decelerationinduced brain injury was hemorrhagic brain contusion.
颅脑减速伤的病理改变主要为出血性脑挫伤.
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Apoptosis is an important pathophysiological characteristic during hepatic ischemia - reperfusion injury ( HIRI ) .
细胞凋亡是肝缺血 — 再 灌注 损伤重要的病理生理特征.
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The risk factors of in - hospital strokes include surgical operation, previous stroke and some pathophysiological changes.
院内卒中多见于行手术治疗或患有心脏病的住院患者, 多为栓塞所致.
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Result 1613 patients showed abnormal signs in and gall bladder , which coincided with the pathophysiological changes.
结果2032例华支睾吸虫病患者B超 检查,发现肝胆异常征象1613例, 符合疾病的病理生理改变.
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The principal pathophysiological mechanism of acute ischemic coronary syndrome ( AICS ) is atherosclerotic plaque rupture.
急性冠状动脉 综合征 发生的主要病理生理机制系粥样斑块破裂.
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It also discusses anatomical, physiological, and pathophysiological features of the cardiovascular, respiratory, and renal systems.
也讨论了心血管 、 呼吸和肾脏系统的解剖学 、 生理学和病理生理学特点.
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Portal hypertension, splanchnic vasodilatation, liver insufficiency, and cardiovascular dysfunction are major pathophysiological hallmarks.
病理改变包括门静脉高压, 脾功能亢进, 肝功能恶化,心功能下降.
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Diffuse intravascular coagulation ( DIC ) can be the underlying pathophysiological mechanism that leads to spontaneous bleeding.
弥漫性的血管内凝血 ( DIC ) 可能是导致自发出血的病理生理学机制.
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The oxygen - free radical may be involved in the pathophysiological process of acute phase Corpulmonale.
而氧自由基可能参与慢性肺心病急性期的病理生理过程.
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Moreover, it contributes to the elucidation of the pathophysiological mechanisms of atherosclerosis and some metabolic diseases.
同时也为研究动脉粥样硬化和高脂血症等多种代谢疾病的发病机理提供了重要证据.
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However, pain and dysfunction are considered separately because they usually result from different pathophysiological events.
但疼痛及功能障碍是被分开考虑的,因为它们通常起因于不同的病理生理过程.
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Further investigations of the pathophysiological roles of MAPKKK 5 may suggest concrete strategies to overcome refractory TLE.
MAPKKK5的病理生理作用的深入研究可能有利于提出治疗难治性颞叶癫痫的策略.
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