Ras gene mutation in childhood AML was mainly found in M 2 , M 4 , M 5 subtypes.
AML患儿中ras基因突变多见于M2 、 M4及M5型.
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Objective : To study the biological and clinical significance of CD 7 + CD 56 + AML.
目的: 探讨CD7+CD56+急性髓系白血病(AML)的临床生物学意义.
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Of the 30 patients with AML, 21 ( 70 % ) got 4 year DFS.
AML共30例, 无病生存达4年以上者占70% ( 21/30 ).
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Objective : To analyze CT features of angiomyolipoma ( AML ).
目的探讨乏脂肪肾错构瘤 ( angiomyolipoma,AML ) 的螺旋CT表现特征.
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Results There were no difference in LDH and ? ? 2 - MG between AML subtypes patients.
结果AML各型患者的血LDH和β 2-MG 水平相同.
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Thus , the diagnosis of multiple myeloma in remission and secondary AML - M 4 was established.
2继发性急性粒单核细胞白血病(AML -M4 ).
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No significant difference of bone marrow SI value could be seen between ALL and AML ( P & gt ; 0.05 ).
急性淋巴细胞白血病(ALL)同髓细胞白血病 ( AML)骨髓SI值之间的差异无显著性意义(P >0.05 ).
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The expression of CD 133 were highly positive in 2 AML patients transformed from MDS.
2例MDS转化 的AML均为CD133高表达.
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Objective To explore the immunophenotype of children with acute myeloid leukemia ( AML ) and its clinical significance.
目的探讨儿童急性粒细胞白血病 ( AML ) 免疫分型及其临床意义.
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The expression of Fas was detected in 54 patients with AML by flow cytometery.
采用直接免疫荧光法检测54例AML患者Fas抗原的表达情况.
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Acute myeloid leukemia ( AML ) is a clonal and heterogeneous disease characterized by a myriad of genetic defects.
急性髓性白血病 ( acutemyeloid leukemia, AML ) 是一类起源于造血干细胞的、以大量遗传缺陷为特征的异质性恶性血液病.
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Methods: Induction regiment of acute myeloid leukemia ( AML ) consisted of mitoxantrone and cytosine arabinoside ( Ara C ).
方法: 急性髓细胞白血病 ( AML ) 诱导治疗采用MTZ和阿糖胞苷 ( Ara-C ).
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Objective To investigation the cytogenetic characteristic of the childhood acute mye logenous leukemia ( AML ).
目的 了解儿童急性粒细胞白血病 ( AML ) M2亚型遗传学特征.
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Objective To explore the potentiality of retroviral etiology in human acute myeloid leukemia ( AML ).
目的探索人类急性髓系白血病 ( AML ) 逆转录病毒病因学的可能性.
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Conclusion NVP - can inhibit the proliferation and induce the apoptosis of BMMNC of AML patients in vitro.
结论NVP - 可抑制AML患者BMMNC生长并诱导其凋亡.
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