The expression of Fas increased with PYM concentration while the expression of Bcl - 2 decreased.
Fas表达随药物浓度增加而递减,Bcl -2 表达随药物浓度增加而升高.
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Conclusion: QY and YP can lower the expression of Bcl - 2 but not act on P 53.
结论: 清毒饮、养正片确能下调Bcl -2 表达,而对P53作用不够明显.
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Conclusion Bcl - 2 gene family may participate in the DATS induced apoptosis of MGC - 803 cells.
结论Bcl -2 基因家族可能参与了DATS诱导的MGC -803 细胞凋亡.
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The staining of TOPO - II α, P - gp, GST - π and BCL - 2 are no significant different between them.
而TOPO- 、 P-gp 、 GST - π、BCL -2 的表达差异无显著性.
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Conclusion: Narenmandula - 11 plays role in inhibiting tumor, which may be related to upregulation of bcl - 2 protein.
结论: 那仁满都拉-11,有抑制肿瘤的作用, 可能与bcl -2 蛋白上调有关.
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So to a certain extent, bcl - 2 and bax expression might reflect the DG and biological behavior.
推测bcl -2 、bax表达水平在一定程度上反映了肿瘤的生物学行为,可作为预测预后的指标.
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The lev - els of Bcl - 2 and Bcl - X _ L proteins were examined by western blot analysis.
Westernblot法分析细胞内Bcl -2 和Bcl -X _L蛋白水平的变化.
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Conclusion Bcl - 2 , Bax, Bcl - xl proteins might participate in the carcinogenesis and development of BCC.
结论Bcl -2 、 Bcl-xl 、 Bax蛋白可能参与了基底细胞癌的发生、发展.
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The Study on Effect of Apoptosis - related Gene C - myc, bcl - 2 and p 53 in Infancy Hemangiomas.
凋亡相关基因C-myc, bcl -2 ,p53在小儿血管瘤中的作用研究.
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The expression of p 185 was reversely correlated to that of bcl - 2 in ChC.
p185表达与bcl -2 表达呈负相关.
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The lev - els of Bcl - 2 and Bcl - X _ L proteins were examined by western blot analysis.
Westernblot法分析细胞内Bcl -2 和Bcl -X _L蛋白水平的变化.
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Conlusion 1 Over expression of Bcl - 2 was observed in biliary carcinomas.
结论1Bcl -2 蛋白在胆管癌与良性胆管疾病中表达有显著差异,它的过度表达与肝外胆管癌的发生有关.
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Conclusion: Bcl - 2 was restraining apoptosis gene, Bax, Bak were promoting apoptosis gene.
结论: Bcl一2为抑凋亡基因, Bax和 Bak为促凋一亡基因, 它们可以作为评估喉鳞癌生物学特性的参考指标.
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Bcl - 2 protein immunoreactivity was associated with mitochondria and ER.
Bcl2分布与线粒体和内质网有关.
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Fas mRNA was up - regulutedand Bcl - 2 mRNA was down - regulated by YWN.
异位宁促使异位内膜细胞FasmRNA高表达及Bc1-2mRNA低表达.
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